By Dana Bell
Air strength colours Vol.I: 1926-1942
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H. H. Neubert, and A. Heinz. 2012. Does human leukocyte elastase degrade intact skin elastin? FEBS J 279(22): 4191–4200. Schräder, C. , A. Heinz, and C. E. H. Schmelzer. 2016. Unpublished data. Senior, R. , G. L. Griffin, C. J. Fliszar et al. 1991. Human 92- and 72-kilodalton type IV collagenases are elastases. J Biol Chem 266(12): 7870–7875. Shapiro, S. D. 2002. Neutrophil elastase: Path clearer, pathogen killer, or just pathologic? Am J Respir Cell Mol Biol 26(3): 266–268. Assembly and Properties of Elastic Fibers ◾ 29 Shapiro, S.
1993. Emilin, a component of elastic fibers preferentially located at the elastin-microfibrils interface. J Cell Biol 121(1): 201–212. Broekelmann, T. , B. A. Kozel, H. Ishibashi et al. 2005. Tropoelastin interacts with cell-surface glycosaminoglycans via its COOH-terminal domain. J Biol Chem 280(49): 40939–40947. , T. Broekelmann, J. Rosenbloom, and R. P. Mecham. 1996. Functional domains on elastin and microfibril-associated glycoprotein involved in elastic fibre assembly. Biochem J 318(Pt 1): 149–155.
1986, Novinec et al. 2007). Elastases are produced by fibroblasts, platelets, macrophages, leukocytes, mesenchymal cells, or the pancreas (Shapiro 2002). In addition, many aggressive proteases that are capable of degrading elastin are of microbial origin (Morihara and Tsuzuki 1967). Despite the different catalytic mechanisms, all elastases are relatively nonspecific and have the common preference for cleaving at peptide bonds associated with hydrophobic or aromatic amino acids (Mecham et al. 1997).
Air Force Colors, Vol.I. 1926-1942 by Dana Bell